How common is anetoderma?
The exact incidence of anetoderma is unknown, but secondary anetoderma is probably more common than the primary form. Familial anetoderma is uncommon, with only 12 families reported in the literature.
How do I get rid of anetoderma?
Unfortunately, once the lesions of anetoderma have formed, there is no treatment to get rid of the lesions. If the disease involves a small area, surgical excision of the lesions may be an option.
What skin conditions are caused by stress?
Stress-related skin problems include:
- eczema.
- psoriasis.
- acne.
- rosacea.
- ichthyosis.
- vitiligo.
- hives.
- seborrheic dermatitis.
What causes stress skin?
Have you ever noticed that you break out more when you’re stressed? This is because stress causes your body to make hormones like cortisol, which tells glands in your skin to make more oil. Oily skin is more prone to acne and other skin problems.
What is deep morphea?
Deep morphea encompasses a variety of clinical entities in which inflammation and sclerosis are found in the deep dermis, panniculus, fascia, or superficial muscle.
What is anetoderma and what causes it?
Anetoderma is an uncommon condition in which the elastic tissue in the dermis is lost, resulting in a depression in the skin. It is also known as macular atrophy.
What is the prevalence of atrophoderma of Pasini and Pierini?
Atrophoderma of Pasini and Pierini is rarely reported with only about 100 cases in the medical literature. Atrophoderma of Pasini and Pierini usually first appears in adolescence or early adulthood (second and third decade). Onset in infancy, childhood, and late adult-life has been reported, as has congenital presentation.
What are the clinical entities with depressed lesions or atrophoderma?
Other clinical entities with depressed lesions or atrophoderma include anetoderma, long-standing lupus profundus, eosinophilic fasciitis, lipodystrophy, and atrophoderma of Moulin. Except for atrophoderma of Moulin, these entities can be readily differentiated based on the history, clinical, and histopathologic findings.
How is atrophoderma differentiated from morphea?
Atrophoderma may mimic morphea and is differentiated by the characteristic well-demarcated areas with cliff-drop borders, which are usually seen on the trunk. Progressive systemic sclerosis is rare in childhood and usually presents with severe acral and orofacial sclerosis.3